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Its probable mechanism includes activation of renal AGE-RAGE signaling pathways, decreasing levels of monocyte chemotactic protein-1 (MCP-1) and PKC-A, which consequently reduces the production of slit diaphragm proteins such as nephrin and podocin, thereby mitigating the impact of AGE-mediated DN pathogenesis (Muthenna et al., 2014)

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doi: 10.1016/j.jhep.2010.09.032 196 WeyerCFunahashiTTanakaSHottaKMatsuzawaYPratleyREet al

, hydrophobic) motif on a loop that is positioned between the G2 and G3 loop motifs that has long been thought to serve as a high affinity ( K Zn 10 10 10 11 M 1 ) coordination site for cognate metal (Figure 36)

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